Get a proposal

MIGRAINE · White paper

Migraine Clinical Trials

Approved therapies, the pivotal-trial endpoints they were judged on, the magnitude of benefit — and the drugs that failed their endpoints, and why.

Indication overview

About migraine — and why its trials are hard

Migraine is a common, disabling primary neurological disorder characterized by recurrent attacks of moderate-to-severe, often unilateral pulsating headache lasting 4-72 hours, frequently with nausea, photophobia, phonophobia, and sometimes aura. It most affects adults aged 25-55 and is two-to-three times more common in women, reflecting hormonal influences. The dominant neurovascular model implicates activation of the trigeminovascular system and release of calcitonin gene-related peptide (CGRP), which has transformed therapy. Acute treatment historically relied on triptans (5-HT1B/1D agonists) such as sumatriptan; newer options include ditans (lasmiditan) and gepants (ubrogepant, rimegepant). Prevention now includes anti-CGRP monoclonal antibodies (erenumab, fremanezumab, galcanezumab, eptinezumab), oral gepants (atogepant, rimegepant), onabotulinumtoxinA for chronic migraine, and older agents such as topiramate. Trials measure reduction in monthly migraine days, 2-hour pain freedom, and responder rates, and are challenged by high placebo response and historical CGRP-antagonist hepatotoxicity (telcagepant).

Indication
Migraine
ICD-10-CM
G43.909 — Migraine, unspecified

Approved therapies & pivotal evidence

What's been approved — and by how much it moved the endpoint

Drug (brand)ApprovedSettingPivotal trialPrimary endpointMagnitude of benefit
Sumatriptan (Imitrex) 1991Acute treatment of migraine (first triptan, 5-HT1B/1D agonist)Pivotal triptan trialsHeadache relief at 2 hoursRoughly 50-70% headache relief at 2 hours vs ~25-35% placebo (varies by formulation)
OnabotulinumtoxinA (Botox) 2010Preventive treatment of chronic migraine (>=15 headache days/month)PREEMPT 1 and 2Change in frequency of headache days~ -8.4 headache days vs -6.6 with placebo (pooled PREEMPT)
Erenumab (Aimovig) 2018Preventive treatment of migraine (first FDA-approved anti-CGRP receptor monoclonal antibody)STRIVEChange in monthly migraine days140 mg reduced monthly migraine days by ~3.7 vs ~1.8 with placebo
Ubrogepant (Ubrelvy) 2020Acute treatment of migraine with or without aura (oral CGRP receptor antagonist/gepant)ACHIEVE I and IIPain freedom at 2 hours~19-21% pain-free at 2 hours vs ~12% placebo
Rimegepant (Nurtec ODT) 2020Acute treatment (2020) and preventive treatment (2021) of migraine (gepant)Acute Study 303; preventive Study 3052-hour pain freedom (acute); reduction in monthly migraine days (preventive)~21% pain-free at 2h vs ~11% placebo; preventive ~ -4.3 vs -3.5 monthly migraine days
Atogepant (Qulipta) 2021Preventive treatment of episodic migraine (2021); expanded to chronic migraine (2023) (oral gepant)ADVANCEChange in mean monthly migraine days60 mg reduced monthly migraine days by ~ -4.2 vs -2.5 with placebo

Where trials have failed

Drugs that missed their endpoint — and what contributed

The most instructive lessons in migraine development come from programmes that failed the endpoint that mattered.

Drug / trialEndpoint outcomeWhat contributed
Telcagepant — Phase III (Merck)Development haltedHepatotoxicity (elevated liver enzymes) with regular dosing outweighed efficacy
MK-3207 — Phase II (Merck)DiscontinuedLiver-toxicity signals (delayed hepatic laboratory abnormalities)
BI 44370 TA — Phase II (Boehringer Ingelheim)DiscontinuedLack of efficacy relative to development threshold

Choosing the right endpoint

Primary endpoints that matter in migraine trials

  • Reduction in monthly migraine days — Primary efficacy measure for preventive therapies
  • Pain freedom / pain relief at 2 hours — Standard co-primary endpoints for acute-treatment trials
  • Responder rate (>=50% reduction) — Proportion achieving >=50% decrease in monthly migraine days
  • Most bothersome symptom freedom at 2 hours — Acute-trial co-primary alongside pain freedom (nausea, photophobia, or phonophobia)
  • Patient-reported outcomes (MIDAS, HIT-6) — Assess disability and headache impact on daily life

How iNGENū runs migraine trials

Physician-led design, built for FDA submission

Endpoint & biomarker strategy

Board-certified specialists design endpoints and patient selection aligned to current FDA guidance for this indication.

FDA-ready data

Built to ICH-GCP and 21 CFR 312.120, with direct FDA submission — data accepted by the FDA, EMA and MHRA.

Faster, lower-cost delivery

~4-week ethics via the TGA CTN scheme, up to 43.5% R&D rebate, and 80–90% below US CRO cost.

PDF
Migraine - Understanding the Enigma of a Prevalent Neurological Disorder
The full white paper: approved-product analyses, pivotal endpoints and trial-design strategy. Open-access · no sign-in.
Download white paper

Frequently asked questions

Migraine clinical trials — FAQs

How do CGRP monoclonal antibodies differ from gepants?
Anti-CGRP monoclonal antibodies (erenumab, fremanezumab, galcanezumab, eptinezumab) are large injectable/IV biologics dosed monthly or quarterly for prevention. Gepants (ubrogepant, rimegepant, atogepant) are small oral molecules; some treat acute attacks, and rimegepant and atogepant are also approved for prevention.
Why did early CGRP receptor antagonists like telcagepant fail?
Telcagepant and MK-3207 showed efficacy but caused dose- and duration-dependent liver enzyme elevations. This hepatotoxicity, particularly with the frequent dosing needed for prevention, stopped their development, though it did not derail the later, safer gepants.
What is the main efficacy endpoint in migraine prevention trials?
The reduction in mean monthly migraine (or headache) days over a defined period is the primary endpoint, supported by responder rate (>=50% reduction) and patient-reported disability measures such as MIDAS and HIT-6.

Ready to discuss your migraine trial?

Talk to our physician-led team about an FDA-ready, cost-efficient trial design.

Request a proposal