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Oncology · Clinical trials

Non-Small-Cell Lung Cancer Clinical Trials

Approved therapies, the pivotal-trial endpoints they were judged on, the magnitude of benefit — and the drugs that failed their endpoints, and why.

Indication overview

About non-small-cell lung cancer — and why its trials are hard

Non-small-cell lung cancer (NSCLC) accounts for roughly 85% of lung cancers and has become the flagship example of precision oncology. Treatment now hinges on comprehensive molecular profiling and PD-L1 testing at diagnosis. For tumors with targetable driver mutations, oral targeted therapies deliver deep, durable responses: osimertinib is standard for EGFR-mutant disease (FLAURA), alectinib for ALK rearrangements (ALEX), and agents like sotorasib and adagrasib address the previously undruggable KRAS G12C mutation, while amivantamab targets EGFR exon 20 insertions. For tumors without actionable drivers, immune checkpoint inhibitors reshaped care: pembrolizumab monotherapy for high PD-L1 tumors (KEYNOTE-024) and pembrolizumab plus chemotherapy regardless of histology (KEYNOTE-189 for nonsquamous, KEYNOTE-407 for squamous) are first-line standards. Immunotherapy has also moved into earlier stages as adjuvant, neoadjuvant, and perioperative therapy. These advances have meaningfully extended survival, with subsets of driver-positive and immunotherapy-responsive patients achieving multi-year disease control that was unimaginable in the cytotoxic-only era.

Indication
Non-Small-Cell Lung Cancer
ICD-10-CM
C34.90 — Malignant neoplasm of lung

Approved therapies & pivotal evidence

What's been approved — and by how much it moved the endpoint

Drug (brand)ApprovedSettingPivotal trialPrimary endpointMagnitude of benefit
Osimertinib (Tagrisso)2018 (first-line)First-line EGFR exon 19 del/L858R-mutant advanced NSCLCFLAURA (Phase III)PFS then overall survival vs first-gen EGFR-TKIMedian PFS 18.9 vs 10.2 months; median OS 38.6 vs 31.8 months (OS HR 0.80)
Pembrolizumab (monotherapy) (Keytruda)2016First-line PD-L1 >=50%, no EGFR/ALK alterationKEYNOTE-024 (Phase III)PFS and overall survival vs chemotherapyMedian OS 30.0 vs 14.2 months in long-term follow-up; PFS HR 0.50
Pembrolizumab + chemotherapy (Keytruda)2017-2018First-line metastatic nonsquamous (KEYNOTE-189) and squamous (KEYNOTE-407)KEYNOTE-189 (Phase III)Overall survival vs chemotherapy aloneOS HR ~0.49 (nonsquamous); benefit across PD-L1 levels
Alectinib (Alecensa)2017First-line ALK-positive advanced NSCLCALEX (Phase III)Progression-free survival vs crizotinibPFS HR 0.47; median PFS ~34.8 vs 10.9 months, with strong CNS control
Sotorasib (Lumakras)2021Previously treated KRAS G12C-mutant advanced NSCLC (accelerated approval)CodeBreaK 100 (Phase II)Objective response rateORR ~36%; first approved KRAS G12C inhibitor
Amivantamab (Rybrevant)2021EGFR exon 20 insertion-mutant NSCLC after platinum (accelerated approval)CHRYSALIS (Phase I)Objective response rateORR ~40%; first approved therapy for EGFR exon 20 insertions

Where trials have failed

Drugs that missed their endpoint — and what contributed

The most instructive lessons in non-small-cell lung cancer development come from programmes that failed the endpoint that mattered.

Drug / trialEndpoint outcomeWhat contributed
Durvalumab +/- tremelimumab — MYSTIC (Phase III)Failed to meet primary endpoints in first-line metastatic NSCLCDurvalumab did not significantly improve OS over chemotherapy in PD-L1 >=25% patients, and the durvalumab-tremelimumab combination missed its OS and PFS endpoints
Pembrolizumab + ipilimumab — KEYNOTE-598 (Phase III)Failed; no benefit over pembrolizumab aloneAdding ipilimumab to pembrolizumab in PD-L1 >=50% NSCLC did not improve OS or PFS and increased toxicity, so the trial was stopped for futility
Sotorasib (second-line, confirmatory) — CodeBreaK 200 (Phase III)Only modest PFS benefit vs docetaxel; no OS improvementMet PFS but the magnitude was limited and OS was not significantly improved, tempering enthusiasm for first-generation KRAS G12C inhibitors

Choosing the right endpoint

Primary endpoints that matter in non-small-cell lung cancer trials

  • Overall survival (OS) — The definitive endpoint; immunotherapy regimens (KEYNOTE-024/189) and osimertinib demonstrated clear OS gains.
  • Progression-free survival (PFS) — The workhorse endpoint for targeted therapies, where deep responses produce dramatic PFS separation (e.g., ALEX, FLAURA).
  • Objective response rate (ORR) — Basis for accelerated approvals of driver-targeted agents (sotorasib, amivantamab) in biomarker-selected populations.
  • CNS/intracranial activity — A key differentiator for TKIs given the high brain-metastasis rate; osimertinib and alectinib show strong CNS penetration.
  • PD-L1 expression / biomarker selection — PD-L1 tumor proportion score and driver-mutation status determine first-line eligibility and regimen choice.

How iNGENū runs non-small-cell lung cancer trials

Physician-led design, built for FDA submission

Endpoint & biomarker strategy

Board-certified specialists design endpoints and patient selection aligned to current FDA guidance for this indication.

FDA-ready data

Built to ICH-GCP and 21 CFR 312.120, with direct FDA submission — data accepted by the FDA, EMA and MHRA.

Faster, lower-cost delivery

~4-week ethics via the TGA CTN scheme, up to 43.5% R&D rebate, and 80–90% below US CRO cost.

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Frequently asked questions

Non-Small-Cell Lung Cancer clinical trials — FAQs

How is first-line treatment chosen in advanced NSCLC?
By molecular and PD-L1 testing. Tumors with actionable drivers (EGFR, ALK, KRAS G12C, ROS1, and others) receive matched targeted therapy such as osimertinib or alectinib. Driver-negative tumors receive immunotherapy, either pembrolizumab alone for high PD-L1 tumors or combined with chemotherapy.
What changed with KRAS-targeted drugs?
KRAS G12C was long considered undruggable. Sotorasib (2021) and adagrasib (2022) became the first approved KRAS G12C inhibitors, offering an oral option for a common mutation. Responses are meaningful but generally less durable than with EGFR or ALK inhibitors, and confirmatory OS benefit has been limited.
Why have some immunotherapy combinations failed?
Not every combination adds value. Trials like MYSTIC (durvalumab plus tremelimumab) and KEYNOTE-598 (pembrolizumab plus ipilimumab) failed to improve survival over their comparators while adding toxicity, showing that intensifying immunotherapy does not automatically improve outcomes.

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