Get a proposal

Haematology-Oncology · Clinical trials

Acute Lymphoblastic Leukaemia (ALL) Clinical Trials

Approved therapies, the pivotal-trial endpoints they were judged on, the magnitude of benefit — and the drugs that failed their endpoints, and why.

Indication overview

About acute lymphoblastic leukaemia (all) — and why its trials are hard

Acute lymphoblastic leukaemia is an aggressive malignancy of immature lymphoid precursors (most commonly B-cell) that flood the marrow and blood, causing rapid marrow failure. It is the commonest childhood cancer, where intensive multi-agent chemotherapy cures the large majority, but outcomes in adults and in relapsed/refractory disease are markedly worse. Risk stratification relies on immunophenotype, cytogenetics/molecular lesions (notably BCR-ABL1 in Philadelphia-chromosome-positive disease) and minimal residual disease (MRD) response, which is the single most powerful prognostic factor. Immunotherapy has reshaped relapsed B-ALL: the CD19xCD3 bispecific T-cell engager blinatumomab (TOWER) and the anti-CD22 antibody-drug conjugate inotuzumab ozogamicin (INO-VATE) both outperform salvage chemotherapy, and the CD19 CAR-T product tisagenlecleucel (ELIANA) produces high remission rates in paediatric and young-adult relapsed disease. For Ph+ ALL, BCR-ABL tyrosine kinase inhibitors, including the third-generation ponatinib, are integral. Allogeneic stem-cell transplant remains an option for high-risk or MRD-persistent patients. CNS prophylaxis is universal, reflecting ALL's propensity for meningeal involvement.

Indication
Acute Lymphoblastic Leukaemia (ALL)
ICD-10-CM
C91.00 — Acute lymphoblastic leukaemia

Approved therapies & pivotal evidence

What's been approved — and by how much it moved the endpoint

Drug (brand)ApprovedSettingPivotal trialPrimary endpointMagnitude of benefit
Blinatumomab (Blincyto)2014 (relapsed/refractory); later MRD+ and frontline consolidationRelapsed/refractory and MRD-positive B-ALLTOWEROverall survivalMedian OS ~7.7 vs 4.0 months vs chemotherapy (CD19xCD3 BiTE)
Inotuzumab ozogamicin (Besponsa)2017Relapsed/refractory B-ALLINO-VATEComplete remission rate / overall survivalCR/CRi ~81% vs ~29% with chemotherapy; higher MRD negativity
Tisagenlecleucel (Kymriah)2017Relapsed/refractory B-ALL up to age 25ELIANAOverall remission rateORR ~81% within 3 months; durable remissions in paediatric/young-adult patients
Ponatinib (Iclusig)2012 (Ph+ leukaemias); 2024 frontline Ph+ ALLPhiladelphia-chromosome-positive ALL, including T315IPACE; PhALLCON (frontline, with chemotherapy)MRD-negative complete remissionPhALLCON showed superior MRD-negative CR vs imatinib in frontline Ph+ ALL
Imatinib (Gleevec)Established for Ph+ ALL (with chemotherapy)Frontline Philadelphia-chromosome-positive ALLCooperative-group chemotherapy-plus-TKI studiesComplete remission / survivalMarkedly improved remission and transplant rates when added to chemotherapy in Ph+ ALL

Where trials have failed

Drugs that missed their endpoint — and what contributed

The most instructive lessons in acute lymphoblastic leukaemia (all) development come from programmes that failed the endpoint that mattered.

Drug / trialEndpoint outcomeWhat contributed
Rituximab (early anti-CD20 attempts in unselected ALL) — Various before CD20-selected useBenefit was limited/inconsistent until restricted to CD20-positive B-ALL (later confirmed in GRAALL-2005)Efficacy depends on CD20 antigen expression, so unselected use showed no clear advantage
Maxi/high-intensity chemotherapy escalation in older adults — Multiple older-adult intensification studiesFailed to improve survival because treatment-related toxicity offset any anti-leukaemic gainPoor tolerance and comorbidity in older patients; helped drive the shift toward immunotherapy-based, lower-intensity approaches

Choosing the right endpoint

Primary endpoints that matter in acute lymphoblastic leukaemia (all) trials

  • Complete remission (CR/CRi) — Morphologic marrow clearance; foundational response measure
  • Minimal residual disease (MRD) — Most powerful prognostic factor; MRD negativity guides transplant and therapy decisions
  • Overall survival (OS) — Primary endpoint in relapsed adult trials such as TOWER
  • Event-free survival (EFS) — Common in paediatric protocols capturing relapse and induction failure
  • Overall/event-free remission rate — Key single-arm endpoint for CAR-T (ELIANA)

How iNGENū runs acute lymphoblastic leukaemia (all) trials

Physician-led design, built for FDA submission

Endpoint & biomarker strategy

Board-certified specialists design endpoints and patient selection aligned to current FDA guidance for this indication.

FDA-ready data

Built to ICH-GCP and 21 CFR 312.120, with direct FDA submission — data accepted by the FDA, EMA and MHRA.

Faster, lower-cost delivery

~4-week ethics via the TGA CTN scheme, up to 43.5% R&D rebate, and 80–90% below US CRO cost.

Planning a acute lymphoblastic leukaemia (all) trial?
Request a fixed-milestone proposal and a tailored endpoint & feasibility summary for this indication.
Request a proposal

Frequently asked questions

Acute Lymphoblastic Leukaemia (ALL) clinical trials — FAQs

Why does ALL outcome differ so much between children and adults?
Children have more favourable biology and tolerate intensive multi-agent chemotherapy better, achieving high cure rates, whereas adults have more adverse genetics (e.g. Ph-like, Ph+) and greater toxicity, yielding poorer outcomes.
What is Philadelphia-chromosome-positive ALL?
It is ALL carrying the BCR-ABL1 fusion; adding tyrosine kinase inhibitors such as imatinib or ponatinib to chemotherapy dramatically improves remission and survival in this subtype.
How has immunotherapy changed relapsed B-ALL?
Blinatumomab, inotuzumab ozogamicin and CD19 CAR-T (tisagenlecleucel) all produce higher remission and MRD-negativity rates than salvage chemotherapy, often serving as a bridge to allogeneic transplant.

Ready to discuss your acute lymphoblastic leukaemia (all) trial?

Talk to our physician-led team about an FDA-ready, cost-efficient trial design.

Request a proposal